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The LV summit is the most common site of idiopathic epicardial LV VA origins. .
The LV summit is the most superior portion of the LV (star, B) and an important . Figure 2 Left ventricular summit premature ventricular complex (PVC). Earliest .
Premature ventricular contractions originating from the left ventricular (LV) summit pose a serious challenge to catheter ablation, as .One of the most challenging ventricular arrhythmias originates from the left ventricular summit .
While early precordial electrocardiographic (ECG) characteristics are useful to differentiate left-sided from the right-sided outflow tract ventricular arrhythmia (OTVA), few patterns predict an origin from the septal margin of . We performed stepwise catheter ablation on the LV-summit PVC origin site . The LV summit is the most common site of idiopathic epicardial LV VA origins. LV summit VAs are most commonly ablated within the GCV or AIVV but sometimes from the epicardial surface more lateral to these venous structures. The LV summit is the most superior portion of the LV (star, B) and an important anatomic landmark as it is the region on the epicardial surface, where the left main coronary artery (LMCA) bifurcates and is recognized as the commonest source of idiopathic epicardial ventricular arrhythmias (VAs). 35 Anatomic landmarks defining the LV summit are .
Figure 2 Left ventricular summit premature ventricular complex (PVC). Earliest activation is recorded in the anterior interventricular vein (AIV) (left panel) and ablation is not limited by coronary anatomy (right panel).
Premature ventricular contractions originating from the left ventricular (LV) summit pose a serious challenge to catheter ablation, as myocardial thickness, epicardial fat, and coronary vessels impede appropriate radiofrequency (RF) energy delivery to the target areas.One of the most challenging ventricular arrhythmias originates from the left ventricular summit (LVS), a triangular epicardial space with the left main bifurcation as its apex. This area accounts for about 14.0% of LV arrhythmias. While early precordial electrocardiographic (ECG) characteristics are useful to differentiate left-sided from the right-sided outflow tract ventricular arrhythmia (OTVA), few patterns predict an origin from the septal margin of the left ventricular (LV) summit.
We performed stepwise catheter ablation on the LV-summit PVC origin site adjacent to severe coronary artery stenosis using a 3D electroanatomic mapping system in a single case. Special precautions should be taken to avoid coronary artery damage during ablation from distal CVS.
The LV summit is the most superior portion of the epicardial LV bounded by the LAD and LCx. This article describes the ECG and electrophysiological features and results of catheter ablation of idiopathic VAs originating from the LV summit.Ablation of ventricular arrhythmias or premature ventricu-lar contractions (PVCs) from the left ventricular (LV) sum-mit remains challenging because of anatomical constraints, including myocardial thickness and the availability of ac-cess (proximity to major coronary arteries, thick epicardium in an epicardial approach).Surgical cryoablation in the LV summit is a viable option for drug-refractory ventricular arrhythmias. Presurgical epicardial mapping can facilitate the surgical procedure by localizing the area of interest to allow for a more limited surgical dissection of the epicardial fat. The LV summit is the most common site of idiopathic epicardial LV VA origins. LV summit VAs are most commonly ablated within the GCV or AIVV but sometimes from the epicardial surface more lateral to these venous structures.
The LV summit is the most superior portion of the LV (star, B) and an important anatomic landmark as it is the region on the epicardial surface, where the left main coronary artery (LMCA) bifurcates and is recognized as the commonest source of idiopathic epicardial ventricular arrhythmias (VAs). 35 Anatomic landmarks defining the LV summit are . Figure 2 Left ventricular summit premature ventricular complex (PVC). Earliest activation is recorded in the anterior interventricular vein (AIV) (left panel) and ablation is not limited by coronary anatomy (right panel). Premature ventricular contractions originating from the left ventricular (LV) summit pose a serious challenge to catheter ablation, as myocardial thickness, epicardial fat, and coronary vessels impede appropriate radiofrequency (RF) energy delivery to the target areas.
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One of the most challenging ventricular arrhythmias originates from the left ventricular summit (LVS), a triangular epicardial space with the left main bifurcation as its apex. This area accounts for about 14.0% of LV arrhythmias. While early precordial electrocardiographic (ECG) characteristics are useful to differentiate left-sided from the right-sided outflow tract ventricular arrhythmia (OTVA), few patterns predict an origin from the septal margin of the left ventricular (LV) summit. We performed stepwise catheter ablation on the LV-summit PVC origin site adjacent to severe coronary artery stenosis using a 3D electroanatomic mapping system in a single case. Special precautions should be taken to avoid coronary artery damage during ablation from distal CVS.The LV summit is the most superior portion of the epicardial LV bounded by the LAD and LCx. This article describes the ECG and electrophysiological features and results of catheter ablation of idiopathic VAs originating from the LV summit.
Ablation of ventricular arrhythmias or premature ventricu-lar contractions (PVCs) from the left ventricular (LV) sum-mit remains challenging because of anatomical constraints, including myocardial thickness and the availability of ac-cess (proximity to major coronary arteries, thick epicardium in an epicardial approach).
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